Amanote Research
Register
Sign In
Studies on the Contribution of C-Fos/Ap-1 to Arthritic Joint Destruction.
Journal of Clinical Investigation
- United States
doi 10.1172/jci119277
Full Text
Open PDF
Abstract
Available in
full text
Categories
Medicine
Date
March 15, 1997
Authors
S Shiozawa
K Shimizu
K Tanaka
K Hino
Publisher
American Society for Clinical Investigation
Related search
A Mechanism of AP-1 Suppression Through Interaction of C-Fos With Lamin a/C
Genes and Development
Genetics
Developmental Biology
Activation of the C-Fos Enhancer by the Erk MAP Kinase Pathway Through Two Sequence Elements: The C-Fos AP-1 and P62TCF Sites
Oncogene
Cancer Research
Genetics
Molecular Biology
Opposing Activities of C-Fos and Fra-2 on AP-1 Regulated Transcriptional Activity in Mouse Keratinocytes Induced to Differentiate by Calcium and Phorbol Esters
Oncogene
Cancer Research
Genetics
Molecular Biology
CHOP Enhancement of Gene Transcription by Interactions With Jun/Fos AP-1 Complex Proteins
Molecular and Cellular Biology
Cell Biology
Molecular Biology
New EMBO Member's Review: Matrix Metalloproteinases in the Adult Brain Physiology: A Link Between C-Fos, AP-1 and Remodeling of Neuronal Connections?
EMBO Journal
Immunology
Molecular Biology
Biochemistry
Microbiology
Neuroscience
Medicine
Genetics
Constitutive Expression of the AP-1 Transcription Factors C-Jun, junD, junB, and C-Fos and the Marginal Zone B-Cell Transcription Factor Notch2 in Splenic Marginal Zone Lymphoma
Journal of Molecular Diagnostics
Forensic Medicine
Pathology
Molecular Medicine
Maf Nuclear Oncoprotein Recognizes Sequences Related to an AP-1 Site and Forms Heterodimers With Both Fos and Jun.
Molecular and Cellular Biology
Cell Biology
Molecular Biology
The Proto-Oncogene C-Fos Increases the Sensitivity of Keratinocytes to Apoptosis
Oncogene
Cancer Research
Genetics
Molecular Biology
Studies on the Antibiotic-Resistant Mutants of C. Diphtheriae (1)
Journal of Nippon Medical School
