Targeting Glutamatergic and Cellular Prion Protein Mechanisms of Amyloid Β-Mediated Persistent Synaptic Plasticity Disruption: Longitudinal Studies

Neuropharmacology - United Kingdom
doi 10.1016/j.neuropharm.2017.03.036
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Elsevier BV

Related search

Synaptic Plasticity Disruption by Amyloid Β Protein: Modulation by Potential Alzheimer's Disease Modifying Therapies: Figure 1

Biochemical Society Transactions
Biochemistry
2005English

Β-Amyloid 42; Prion Protein (PRNP; PrP; CD230)

Science-Business eXchange
2012English

Molecular Mechanisms for Simvastatin-Mediated Enhancement of Synaptic Plasticity

Molecular Neurodegeneration
Molecular NeuroscienceNeurologyMolecular BiologyCellular
2012English

Cellular Mechanisms for Amyloid Β-Protein Activation of Rat Cholinergic Basal Forebrain Neurons

Journal of Neurophysiology
NeurosciencePhysiology
2001English

Amyloid-Β Receptors: The Good, the Bad, and the Prion Protein

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2015English

Amyloid-Β Nanotubes Are Associated With Prion Protein-Dependent Synaptotoxicity

Nature Communications
AstronomyGeneticsMolecular BiologyBiochemistryChemistryPhysics
2013English

DNA Converts Cellular Prion Protein Into the Β-Sheet Conformation and Inhibits Prion Peptide Aggregation

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2001English

Unraveling Mechanisms of Homeostatic Synaptic Plasticity

Neuron
Neuroscience
2010English

Kinetic Studies of Amyloid Β-Protein Fibril Assembly

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2002English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy