(PDF) Benfotiamine Accelerates the Healing of Ischaemic

Benfotiamine Accelerates the Healing of Ischaemic Diabetic Limbs in Mice Through Protein Kinase B/Akt-Mediated Potentiation of Angiogenesis and Inhibition of Apoptosis

Diabetologia - Germany

doi 10.1007/s00125-005-0103-5

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January 17, 2006

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Springer Nature

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Benfotiamine Accelerates the Healing of Ischaemic Diabetic Limbs in Mice Through Protein Kinase B/Akt-Mediated Potentiation of Angiogenesis and Inhibition of Apoptosis

Diabetologia - Germany

doi 10.1007/s00125-005-0103-5

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

NF-?B Inhibition Accelerates Apoptosis of Bovine Neutrophils

Protein Kinase B (Akt) Promotes Pathological Angiogenesis in Murine Model of Oxygen-Induced Retinopathy

Deguelin, an Akt Inhibitor, Suppresses I B Kinase Activation Leading to Suppression of NF- B-Regulated Gene Expression, Potentiation of Apoptosis, and Inhibition of Cellular Invasion

Multidomain Peptide Hydrogel Accelerates Healing of Full-Thickness Wounds in Diabetic Mice

GqProtein-induced Apoptosis Is Mediated by AKT Kinase Inhibition That Leads to Protein Kinase C-Induced C-Jun N-Terminal Kinase Activation

Insulin Signaling Through Akt/Protein Kinase B Analyzed by Small Interfering RNA-mediated Gene Silencing

Dominant Inhibition of Akt/Protein Kinase B Signaling by the Matrix Protein of a Negative-Strand RNA Virus

Topical Vascular Endothelial Growth Factor Accelerates Diabetic Wound Healing Through Increased Angiogenesis and by Mobilizing and Recruiting Bone Marrow-Derived Cells

Rosiglitazone Accelerates Wound Healing by Improving Endothelial Precursor Cell Function and Angiogenesis in Db/Db Mice