Fibroblast Growth Factor 22 Inhibits ER Stress-Induced Apoptosis and Improves Recovery of Spinal Cord Injury

Frontiers in Pharmacology - Switzerland
doi 10.3389/fphar.2020.00018
Full Text
Abstract

Available in full text

Categories
Pharmacology
Date
Authors
Publisher

Frontiers Media SA

Related search

CD36 Deletion Improves Recovery From Spinal Cord Injury

Experimental Neurology
Developmental NeuroscienceNeurology
2014English

Sustained Expression of Vascular Endothelial Growth Factor and Angiopoietin-1 Improves Blood–Spinal Cord Barrier Integrity and Functional Recovery After Spinal Cord Injury

Journal of Neurotrauma
Neurology
2010English

Inhibiting Epidermal Growth Factor Receptor Improves Structural, Locomotor, Sensory, and Bladder Recovery From Experimental Spinal Cord Injury

Journal of Neuroscience
Neuroscience
2007English

Vascular Endothelial Growth Factor and Spinal Cord Injury Pain

Journal of Neurotrauma
Neurology
2010English

The Nociceptin Receptor Inhibits Axonal Regeneration and Recovery From Spinal Cord Injury

Science Signaling
BiochemistryCell BiologyMolecular Biology
2018English

Elongation Factor-1 Alpha Is a Selective Regulator of Growth Factor Withdrawal and ER Stress-Induced Apoptosis

Cell Death and Differentiation
Cell BiologyMolecular Biology
2002English

The Immunomodulator Decoy Receptor 3 Improves Locomotor Functional Recovery After Spinal Cord Injury

Journal of Neuroinflammation
Molecular NeuroscienceNeurologyImmunologyCellularNeuroscience
2016English

Fibroblast Growth Factor 1 Ameliorates Diabetes-Induced Liver Injury by Reducing Cellular Stress and Restoring Autophagy

Frontiers in Pharmacology
Pharmacology
2020English

Maladaptive Spinal Plasticity Opposes Spinal Learning and Recovery in Spinal Cord Injury

Frontiers in Physiology
Physiology
2012English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy