Piper Sarmentosum Promotes Endothelial Nitric Oxide Production by Reducing Asymmetric Dimethylarginine in Tumor Necrosis Factor-Α-Induced Human Umbilical Vein Endothelial Cells

Frontiers in Pharmacology- Switzerland

doi 10.3389/fphar.2019.01033

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September 17, 2019

Authors

Uma Mahgesswary Sundar

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Frontiers Media SA

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Piper Sarmentosum Promotes Endothelial Nitric Oxide Production by Reducing Asymmetric Dimethylarginine in Tumor Necrosis Factor-Α-Induced Human Umbilical Vein Endothelial Cells

Frontiers in Pharmacology- Switzerland

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Effect of Caffeic Acid on Tumor Necrosis Factor-Alpha-Induced Vascular Inflammation in Human Umbilical Vein Endothelial Cells

Shear-Induced Nitric Oxide Production by Endothelial Cells

Detection of Inflammatory Circulating Endothelial Cells Using Human Umbilical Vein Endothelial Cells Detached From Culture Dishes by Tumor Necrosis Factor-Alpha as Control Cells

Asymmetric Dimethylarginine, Endothelial Nitric Oxide Bioavailability and Mortality in Sepsis

Regulation of Nitric Oxide Synthesis by Proinflammatory Cytokines in Human Umbilical Vein Endothelial Cells. Elevations in Tetrahydrobiopterin Levels Enhance Endothelial Nitric Oxide Synthase Specific Activity.

Phosphatidylinositol Inhibits Vascular Endothelial Growth Factor-A– Induced Migration of Human Umbilical Vein Endothelial Cells

Effect of Interleukin-10 and Laminar Shear Stress on Endothelial Nitric Oxide Synthase and Nitric Oxide in African American Human Umbilical Vein Endothelial Cells

Adiponectin-Induced Endothelial Nitric Oxide Synthase Activation and Nitric Oxide Production Are Mediated by APPL1 in Endothelial Cells

C5a-Induced Gene Expression in Human Umbilical Vein Endothelial Cells