MSP-RON Signaling Is Activated in the Transition From Pancreatic Intraepithelial Neoplasia (PanIN) to Pancreatic Ductal Adenocarcinoma (PDAC)

Frontiers in Physiology - Switzerland
doi 10.3389/fphys.2019.00147
Full Text
Abstract

Available in full text

Categories
Physiology
Date
Authors
Publisher

Frontiers Media SA

Related search

Overexpression of Mesothelin in Pancreatic Ductal Adenocarcinoma (PDAC)

International Journal of Medical Sciences
Medicine
2020English

A Thrombin-Activated PAR-1 Pathway Drives Pancreatic Ductal Adenocarcinoma (PDAC) Growth and Metastasis

Journal of Purdue Undergraduate Research
2019English

Clinicopathological Correlates of Pancreatic Intraepithelial Neoplasia: A Comparative Analysis of 82 Cases With and 152 Cases Without Pancreatic Ductal Adenocarcinoma

Modern Pathology
Forensic MedicinePathology
2003English

Multicomponent Analysis of the Pancreatic Adenocarcinoma Progression Model Using a Pancreatic Intraepithelial Neoplasia Tissue Microarray

Modern Pathology
Forensic MedicinePathology
2003English

Genomic and Transcriptomic Characterization of Advanced Pancreatic Ductal Adenocarcinoma (PDAC) May Inform Systemic Therapy

HPB
HepatologyGastroenterology
2017English

Pancreatic Ductal Adenocarcinoma With Cystic Features

HPB
HepatologyGastroenterology
2016English

Non-Coding RNAs in Pancreatic Ductal Adenocarcinoma

Frontiers in Oncology
Cancer ResearchOncology
2020English

Tumor-Stroma Interactions in Pancreatic Ductal Adenocarcinoma

Molecular Cancer Therapeutics
Cancer ResearchOncology
2007English

Metformin Restrains Pancreatic Duodenal Homeobox-1 (PDX-1) Function by Inhibiting ERK Signaling in Pancreatic Ductal Adenocarcinoma.

Current Molecular Medicine
BiochemistryMedicineMolecular MedicineMolecular Biology
2016English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2025 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy