(PDF) Post-Translational Modifications of the Tax

Post-Translational Modifications of the Tax Oncoprotein of Human T-Cell Leukemia Virus Control Cytoplasmic and Nuclear Steps in Tax-Mediated Activation of the NF-κB Pathway

Retrovirology - United Kingdom

doi 10.1186/1742-4690-6-s2-o17

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September 1, 2009

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Springer Science and Business Media LLC

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Amanote Research

Post-Translational Modifications of the Tax Oncoprotein of Human T-Cell Leukemia Virus Control Cytoplasmic and Nuclear Steps in Tax-Mediated Activation of the NF-κB Pathway

Retrovirology - United Kingdom

doi 10.1186/1742-4690-6-s2-o17

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Akt Activation by Human T-Cell Leukemia Virus Tax Oncoprotein

Kinetic Analysis of Human T-Cell Leukemia Virus Type I Tax-Mediated Activation of NF-kappa B.

Human T-Cell Leukemia Virus Type I Tax Down-Regulates the Expression of Phosphatidylinositol 3,4,5-Trisphosphate Inositol Phosphatases via the NF-κB Pathway.

Bromodomain and Extraterminal (BET) Protein Inhibition Suppresses Human T Cell Leukemia Virus 1 (HTLV-1) Tax Protein-Mediated Tumorigenesis by Inhibiting Nuclear Factor κB (NF-κB) Signaling

Activation of NF- B by the Human T Cell Leukemia Virus Type I Tax Oncoprotein Is Associated With Ubiquitin-Dependent Relocalization of I B Kinase

Genomic Instability Driven by the Human T-Cell Leukemia Virus Type I (HTLV-I) Oncoprotein, Tax

A Human T-Cell Leukemia Virus Tax Variant Incapable of Activating NF-κB Retains Its Immortalizing Potential for Primary T-Lymphocytes

Constitutive Dephosphorylation and Activation of a Member of the Nuclear Factor of Activated T Cells, NF-AT1, in Tax-Expressing and Type I Human T-Cell Leukemia Virus-Infected Human T Cells

Clastogenic Effect of the Human T-Cell Leukemia Virus Type I Tax Oncoprotein Correlates With Unstabilized DNA Breaks