The Viable Mycobacterium Tuberculosis H37Ra Strain Induces a Stronger Mouse Macrophage Response Compared to the Heat-Inactivated H37Rv Strain

Molecular Medicine Reports - Greece
doi 10.3892/mmr.2013.1363
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Spandidos Publications

Related search

Genomic Analysis Reveals Variation Between Mycobacterium Tuberculosis H37Rv and the Attenuated M. Tuberculosis H37Ra Strain

Infection and Immunity
ParasitologyInfectious DiseasesImmunologyMicrobiology
2000English

Mycobacterium Tuberculosis Strain H37rv Infection Towards Matrix Metalloproteinase (Mmp)-2 in Brain

MNJ (Malang Neurology Journal)
2017English

Mycobacterium Tuberculosis Heat-Shock Protein 16.3 Induces Macrophage M2 Polarization Through CCRL2/CX3CR1

Inflammation
AllergyImmunology
2019English

Transcriptionally Induced Enhancers in the Macrophage Immune Response to Mycobacterium Tuberculosis Infection

BMC Genomics
BiotechnologyGenetics
2019English

Complete Genome Sequence of Mycobacterium Tuberculosis DKC2, the Predominant Danish Outbreak Strain

Microbiology Resource Announcements
ImmunologyGeneticsMolecular BiologyMicrobiology
2019English

Drug Resistance Evolution of a Mycobacterium Tuberculosis Strain From a Noncompliant Patient

Journal of Clinical Microbiology
Microbiology
2005English

Rapid Susceptibility Testing of Mycobacterium Tuberculosis (H37Ra) by Flow Cytometry.

Journal of Clinical Microbiology
Microbiology
1995English

A Diabetic Strain of the Mouse

Proceedings of the Japan Academy
1962English

Macrophage Migration Inhibitory Factor (MIF) Is a Critical Mediator of the Innate Immune Response to Mycobacterium Tuberculosis

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
2013English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2025 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy