PTPN14 Degradation by High-Risk Human Papillomavirus E7 Limits Keratinocyte Differentiation and Contributes to HPV-mediated Oncogenesis

Proceedings of the National Academy of Sciences of the United States of America- United States

doi 10.1073/pnas.1819534116

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March 20, 2019

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Proceedings of the National Academy of Sciences

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PTPN14 Degradation by High-Risk Human Papillomavirus E7 Limits Keratinocyte Differentiation and Contributes to HPV-mediated Oncogenesis

Proceedings of the National Academy of Sciences of the United States of America- United States

Full Text

Abstract

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Date

Authors

Publisher

Related search

The PTPN14 Tumor Suppressor Is a Degradation Target of Human Papillomavirus E7

The Human Papillomavirus (HPV) E7 Protein Antagonises an Imiquimod-Induced Inflammatory Pathway in Primary Human Keratinocytes

DNA-EIA to Detect High and Low Risk HPV Genotypes in Cervical Lesions With E6/E7 Primer Mediated Multiplex PCR

Screening for Oral High-Risk Human Papillomavirus (HPV) Among a Pediatric Patient Population

Human Scribble (Vartul) Is Targeted for Ubiquitin-Mediated Degradation by the High-Risk Papillomavirus E6 Proteins and the E6AP Ubiquitin-Protein Ligase

Comparison of the Abbott RealTime High-Risk Human Papillomavirus (HPV), Roche Cobas HPV, and Hybrid Capture 2 Assays to Direct Sequencing and Genotyping of HPV DNA

Screening of Novel Inhibitors Targeting Human Papillomavirus 16 E6/Ap/P53 Ternary Complex Towards Development of Therapeutic Strategies Against HPV-mediated Oncogenesis

High-Risk Human Papillomavirus E6/E7 mRNA and L1 DNA as Markers of Residual/Recurrent Cervical Intraepithelial Neoplasia

Human Papillomavirus (HPV) Infection and Immunohistochemistry