(PDF) Early-Onset Type 2 Diabetes in Obese White Subjects

Early-Onset Type 2 Diabetes in Obese White Subjects Is Characterised by a Marked Defect in Beta Cell Insulin Secretion, Severe Insulin Resistance and a Lack of Response to Aerobic Exercise Training

Diabetologia - Germany

doi 10.1007/s00125-007-0655-7

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March 29, 2007

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Springer Science and Business Media LLC

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Early-Onset Type 2 Diabetes in Obese White Subjects Is Characterised by a Marked Defect in Beta Cell Insulin Secretion, Severe Insulin Resistance and a Lack of Response to Aerobic Exercise Training

Diabetologia - Germany

doi 10.1007/s00125-007-0655-7

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Impairment of Early Insulin Response After Glucose Load, Rather Than Insulin Resistance, Is Responsible for Postprandial Hyperglycemia Seen in Obese Type 2 Diabetes. Assessment Using Nateglinide, a New Insulin Secretagogue.

Vinegar Improves Insulin Sensitivity to a High-Carbohydrate Meal in Subjects With Insulin Resistance or Type 2 Diabetes

Different Aetiologies of Type 2 (Non-Insulin-Dependent) Diabetes Mellitus in Obese and Non-Obese Subjects

Changes in Plasma Visfatin and Insulin Resistance Index in Obese Women With Type 2 Diabetes After Pilates Exercise

PCLO Variants Are Nominally Associated With Early-Onset Type 2 Diabetes and Insulin Resistance in Pima Indians

Acute Lowering of Circulating Fatty Acids Improves Insulin Secretion in a Subset of Type 2 Diabetes Subjects

Cell Type–specific Immune Phenotypes Predict Loss of Insulin Secretion in New-Onset Type 1 Diabetes

SIRT5 Regulates Pancreatic Β‑cell Proliferation and Insulin Secretion in Type 2 Diabetes

HOMA-beta in the UKPDS and ADOPT. Is the Natural History of Type 2 Diabetes Characterised by a Progressive and Inexorable Loss of Insulin Secretory Function? Maybe? Maybe Not?