Non-Canonical Caspase-1 Signaling Drives RIP2-Dependent and TNF-α-Mediated Inflammation in Vivo

Cell Reports- United States

doi 10.1016/j.celrep.2020.01.090

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February 1, 2020

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Elsevier BV

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Non-Canonical Caspase-1 Signaling Drives RIP2-Dependent and TNF-α-Mediated Inflammation in Vivo

Cell Reports- United States

Full Text

Abstract

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Date

Authors

Publisher

Related search

Functional Crosstalk Between Non‐canonical Caspase‐11 and Canonical NLRP3 Inflammasomes During Infection‐mediated Inflammation

Extracellular Caspase-6 Drives Murine Inflammatory Pain via Microglial TNF-α Secretion

Mesenchymal Stromal Cells Directly Promote Inflammation by Canonical NLRP3 and Non-Canonical Caspase-11 Inflammasomes

Overexpression of HO-1 Protects Against TNF-α-Mediated Airway Inflammation by Down-Regulation of TNFR1-Dependent Oxidative Stress

C-Flip Protects Eosinophils From TNF-α-Mediated Cell Death in Vivo

TNF Induces Caspase-Dependent Inflammation in Renal Endothelial Cells Through a Rho- And Myosin Light Chain Kinase-Dependent Mechanism

Caspase-11 Non-Canonical Inflammasome: Emerging Activator and Regulator of Infection-Mediated Inflammatory Responses

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Caspase-8 Regulates TNF-α-induced Epithelial Necroptosis and Terminal Ileitis