T-Cell Tropism and the Role of ORF66 Protein in Pathogenesis of Varicella-Zoster Virus Infection

Journal of Virology - United States
doi 10.1128/jvi.79.20.12921-12933.2005
Full Text
Abstract

Available in full text

Date
Authors
Publisher

American Society for Microbiology

Related search

Varicella-Zoster Virus T Cell Tropism and the Pathogenesis of Skin Infection

Current Topics in Microbiology and Immunology
AllergyImmunologyMicrobiology
2010English

The ORF47 and ORF66 Putative Protein Kinases of Varicella-Zoster Virus Determine Tropism for Human T Cells and Skin in the SCID-hu Mouse

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
1998English

The Varicella-Zoster Virus ORF66 Protein Induces Kinase Activity and Is Dispensable for Viral Replication.

Journal of Virology
Insect ScienceImmunologyMicrobiologyVirology
1996English

Egress of Varicella-Zoster Virus From the Melanoma Cell: A Tropism for the Melanocyte.

Journal of Virology
Insect ScienceImmunologyMicrobiologyVirology
1995English

Clinical Features of Varicella-Zoster Virus Infection

Viruses
VirologyInfectious Diseases
2018English

Varicella Zoster Virus Specific T Cell Responses in Untreated Giant Cell Arteritis

Arthritis and Rheumatology
RheumatologyAllergyImmunology
2017English

Clinical and Molecular Aspects of Varicella Zoster Virus Infection

Future Neurology
Neurology
2009English

Seroepidemiology of Varicella Zoster Virus Infection in Vojvodina, Serbia

Epidemiology and Infection
EpidemiologyInfectious Diseases
2018English

Congenital Varicella-Zoster Virus Infection and Barrett's Esophagus

Journal of Infectious Diseases
Infectious DiseasesAllergyImmunology
1998English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2025 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy