Withdrawal: Cooperative Regulation of NOTCH1 Protein-Phosphatidylinositol 3-Kinase (PI3K) Signaling by NOD1, NOD2, and TLR2 Receptors Renders Enhanced Refractoriness to Transforming Growth Factor-Β (TGF-β)- Or Cytotoxic T-Lymphocyte Antigen 4 (CTLA-4)-mediated Impairment of Human Dendritic Cell Maturation.

Journal of Biological Chemistry - United States
doi 10.1074/jbc.w119.011943
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American Society for Biochemistry & Molecular Biology (ASBMB)

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