Distinct Licensing of IL-18 and IL-1β Secretion in Response to NLRP3 Inflammasome Activation

PLoS ONE- United States

doi 10.1371/journal.pone.0045186

Full Text

Abstract

Available in

Categories

Multidisciplinary

Date

September 18, 2012

Authors

Rebecca L. Schmidt

Publisher

Public Library of Science (PLoS)

Related search

Chlamydial Infection of Monocytes Stimulates IL-1β Secretion Through Activation of the NLRP3 Inflammasome

Microbes and Infection

Infectious Diseases

English

Neutrophilic NLRP3 Inflammasome-Dependent IL-1β Secretion Regulates the γδT17 Cell Response in Respiratory Bacterial Infections

Mucosal Immunology

English

Protein-Bound Polysaccharide-K Induces IL-1β via TLR2 and NLRP3 Inflammasome Activation

Innate Immunity

Infectious Diseases

Molecular Biology

English

IL-1β Activation in Response to Staphylococcus Aureus Lung Infection Requires Inflammasome-Dependent and Independent Mechanisms

European Journal of Immunology

English

The NLRP3 Inflammasome and IL-1β Accelerate Immunologically Mediated Pathology in Experimental Viral Fulminant Hepatitis

Molecular Biology

English

Only IL-1β Release Is Inflammasome-Dependent Upon Ultraviolet B Irradiation Although IL-18 Is Also Secreted

Molecular Biology

English

Glucosamine Inhibits IL-1β Expression by Preserving Mitochondrial Integrity and Disrupting Assembly of the NLRP3 Inflammasome

Scientific Reports

Multidisciplinary

English

The NLRP3 Inflammasome Modulates Glycolysis by Increasing PFKFB3 in an IL-1β-dependent Manner in Macrophages

Scientific Reports

Multidisciplinary

English

L5-LDL From ST-elevation Myocardial Infarction Patients Induces IL-1β Production via LOX-1 and NLRP3 Inflammasome Activation in Macrophages

American Journal of Physiology - Heart and Circulatory Physiology

Cardiovascular Medicine

English