(PDF) Stimulation of Tat-Associated Kinase-Independent

Stimulation of Tat-Associated Kinase-Independent Transcriptional Elongation From the Human Immunodeficiency Virus Type-1 Long Terminal Repeat by a Cellular Enhancer

EMBO Journal - Germany

doi 10.1093/emboj/18.5.1378

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Date

March 1, 1999

Authors

M. J. West

Publisher

Wiley

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Stimulation of Tat-Associated Kinase-Independent Transcriptional Elongation From the Human Immunodeficiency Virus Type-1 Long Terminal Repeat by a Cellular Enhancer

EMBO Journal - Germany

doi 10.1093/emboj/18.5.1378

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Interactions of Thyroid Hormone Receptor With the Human Immunodeficiency Virus Type 1 (HIV-1) Long Terminal Repeat and the HIV-1 Tat Transactivator.

Repression in Vitro, by Human Adenovirus E1A Protein Domains, of Basal or Tat-Activated Transcription of the Human Immunodeficiency Virus Type 1 Long Terminal Repeat.

Interleukin-6 and Glucocorticoids Synergistically Induce Human Immunodeficiency Virus Type-1 Expression in Chronically Infected U1 Cells by a Long Terminal Repeat Independent Post-Transcriptional Mechanism

Repression of Human Immunodeficiency Virus Type 1 Long Terminal Repeat-Driven Gene Expression by Binding of the Virus to Its Primary Cellular Receptor, the CD4 Molecule.

Structure, Sequence, and Position of the Stem-Loop in Tar Determine Transcriptional Elongation by Tat Through the HIV-1 Long Terminal Repeat.

The Human Factors YY1 and LSF Repress the Human Immunodeficiency Virus Type 1 Long Terminal Repeat via Recruitment of Histone Deacetylase 1

Molecular and Functional Interactions of Transcription Factor USF With the Long Terminal Repeat of Human Immunodeficiency Virus Type 1.

Evidence for Neurotoxic Activity of Tat From Human Immunodeficiency Virus Type 1

Murine Leukemia Virus-Based Tat-Inducible Long Terminal Repeat Replacement Vectors: A New System for Anti-Human Immunodeficiency Virus Gene Therapy.