(PDF) Epigenetically Repressing Human Cytomegalovirus Lytic

Epigenetically Repressing Human Cytomegalovirus Lytic Infection and Reactivation From Latency in THP-1 Model by Targeting H3K9 and H3K27 Histone Demethylases

PLoS ONE - United States

doi 10.1371/journal.pone.0175390

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April 13, 2017

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Amanote Research

Epigenetically Repressing Human Cytomegalovirus Lytic Infection and Reactivation From Latency in THP-1 Model by Targeting H3K9 and H3K27 Histone Demethylases

PLoS ONE - United States

doi 10.1371/journal.pone.0175390

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Involvement of Histone H3 Lysine 9 (H3K9) Methyltransferase G9a in the Maintenance of HIV-1 Latency and Its Reactivation by BIX01294

Sifting and Winnowing Through Human Cytomegalovirus Lytic Replication and Latency

Human Cytomegalovirus Latency and Reactivation in Allogeneic Hematopoietic Stem Cell Transplant Recipients

Targeting Histone Demethylases in Cancer Therapy

Quiescence Promotes Latent HIV Infection and Resistance to Reactivation From Latency With Histone Deacetylase Inhibitors

Human Cytomegalovirus Latency: Targeting Differences in the Latently Infected Cell With a View to Clearing Latent Infection

Inhibition of H3K27me3-Specific Histone Demethylases JMJD3 and UTX Blocks Reactivation of Herpes Simplex Virus 1 in Trigeminal Ganglion Neurons

Direct and Quantitative Single-Cell Analysis of Human Immunodeficiency Virus Type 1 Reactivation From Latency

Gene Expression During Reactivation of Herpes Simplex Virus Type 1 From Latency in the Peripheral Nervous System Is Different From That During Lytic Infection of Tissue Cultures.