Combined Blockade of Interleukin-1 Α and Β Signaling Protects Mice From Cognitive Dysfunction After Traumatic Brain Injury

eNeuro - United States
doi 10.1523/eneuro.0385-17.2018
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Society for Neuroscience

Related search

Blockade of Nociceptin Signaling Reduces Biochemical, Structural and Cognitive Deficits After Traumatic Brain Injury

2010English

Chronic Cerebrovascular Dysfunction After Traumatic Brain Injury

Journal of Neuroscience Research
Molecular NeuroscienceCellular
2016English

Chronic Visual Dysfunction After Blast-Induced Mild Traumatic Brain Injury

Journal of Rehabilitation Research and Development
Rehabilitation
2014English

Preliminary Report on Cardiac Dysfunction After Isolated Traumatic Brain Injury*

Critical Care Medicine
Critical CareIntensive Care Medicine
2014English

Cognitive Predictors of Academic Achievement in Young Children 1 Year After Traumatic Brain Injury.

Neuropsychology
Physiological PsychologyNeuropsychology
2012English

Interleukin 6 Mediates Neuroinflammation and Motor Coordination Deficits After Mild Traumatic Brain Injury and Brief Hypoxia in Mice

Shock
Critical CareEmergency MedicineIntensive Care Medicine
2013English

Sensorintegrative Dysfunction Underlying Vestibular Disorders After Traumatic Brain Injury: A Review

Journal of Rehabilitation Research and Development
Rehabilitation
2012English

Genetic Deletion and Pharmacological Inhibition of Nogo-66 Receptor Impairs Cognitive Outcome After Traumatic Brain Injury in Mice

Journal of Neurotrauma
Neurology
2010English

Hypopituitarism After Traumatic Brain Injury

Cureus
2019English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy