Age Dependent Trans-Cellular Propagation of Human Tau Aggregates in Drosophila Disease Models

doi 10.1101/2020.03.28.013136
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Cold Spring Harbor Laboratory

Related search

Novel Sensors for Detecting Alzheimer's Disease Related Tau Protein Aggregates

Biophysical Journal
Biophysics
2019English

Corrigendum: Genetic Background Influences the Propagation of Tau Pathology in Transgenic Rodent Models of Tauopathy

Frontiers in Aging Neuroscience
AgingCognitive Neuroscience
2020English

Age-Dependent Changes in Transcription Factor FOXO Targeting in Female Drosophila

Frontiers in Genetics
GeneticsMolecular Medicine
2019English

Macroautophagy Deficiency Mediates Age-Dependent Neurodegeneration Through a Phospho-Tau Pathway

Molecular Neurodegeneration
Molecular NeuroscienceNeurologyMolecular BiologyCellular
2012English

Amyloid‐β Oligomers in Cellular Models of Alzheimer’s Disease

Journal of Neurochemistry
BiochemistryMolecular NeuroscienceCellular
2020English

Transgenic Drosophila Models of Alzheimer’s Disease and Tauopathies

Brain Structure and Function
AnatomyNeuroscienceHistology
2009English

Age-Dependent Recombination Rates in Human Pedigrees

PLoS Genetics
EvolutionEcologyGeneticsMolecular BiologyCancer ResearchSystematicsBehavior
2011English

Analysis of PINCH Function in Drosophila Demonstrates Its Requirement in Integrin-Dependent Cellular Processes

Development (Cambridge)
Developmental BiologyMolecular Biology
2003English

Tau Intricacies in Disease

Nature Reviews Molecular Cell Biology
Cell BiologyMolecular Biology
2020English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy