Inflammatory Stimuli Promote Oxidative Stress in Pancreatic Acinar Cells via Toll-Like Receptor 4/Nuclear Factor-κB Pathway

International Journal of Molecular Medicine- Greece

doi 10.3892/ijmm.2018.3906

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October 1, 2018

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Spandidos Publications

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Inflammatory Stimuli Promote Oxidative Stress in Pancreatic Acinar Cells via Toll-Like Receptor 4/Nuclear Factor-κB Pathway

International Journal of Molecular Medicine- Greece

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Hypoxia Exacerbates Inflammatory Acute Lung Injury via the Toll-Like Receptor 4 Signaling Pathway

Dibromoacetic Acid Induced Hepatotoxicity in Mice Through Oxidative Stress and Toll-Like Receptor 4 Signaling Pathway Activation

MicroRNA‑451 Relieves Inflammation in Cerebral Ischemia‑reperfusion via the Toll‑like Receptor 4/MyD88/NF‑κB Signaling Pathway

Nuclear Factor-κB Modulates Cellular Glutathione and Prevents Oxidative Stress in Cancer Cells

MicroRNA-421 Improves Ischemia/Reperfusion Injury via Regulation Toll-Like Receptor 4 Pathway

Toll-Like Receptors Promote Mitochondrial Translocation of Nuclear Transcription Factor Nuclear Factor of Activated T-Cells in Prolonged Microglial Activation

A Standardized Traditional Chinese Medicine Preparation Named Yejuhua Capsule Ameliorates Lipopolysaccharide-Induced Acute Lung Injury in Mice via Downregulating Toll-Like Receptor 4/Nuclear Factor-κB

Cyclic Stretch Enhances the Expression of Toll-Like Receptor 4 Gene in Cultured Cardiomyocytes via P38 MAP Kinase and NF-κB Pathway

MP88-06 NUCLEAR FACTOR (NF)-κB SIGNALS PROMOTE UROTHELIAL TUMORIGENESIS THROUGH THE ANDROGEN RECEPTOR (AR) PATHWAY