(PDF) A Naturally Occurring Variation in the Proline-Rich

A Naturally Occurring Variation in the Proline-Rich Region Does Not Attenuate Human Immunodeficiency Virus Type 1 Nef Function

Journal of Virology - United States

doi 10.1128/jvi.78.18.10197-10201.2004

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September 15, 2004

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American Society for Microbiology

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Human Immunodeficiency Virus Type 1 Nef Does Not Alter T-Cell Sensitivity to Antigen-Specific Stimulation.

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Nef-Induced CD4 Downregulation: A Diacidic Sequence in Human Immunodeficiency Virus Type 1 Nef Does Not Function as a Protein Sorting Motif Through Direct Binding to -COP

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Dynamic Evolution of the Human Immunodeficiency Virus Type 1 Pathogenic Factor, Nef

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Human Immunodeficiency Virus Type 1 Nef: Adapting to Intracellular Trafficking Pathways

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Predicting the Impact of Blocking Human Immunodeficiency Virus Type 1 Nef in Vivo

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Human Immunodeficiency Virus Type 1 Nef Induces Accumulation of CD4 in Early Endosomes.

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Proline-Rich Tandem Repeats of Antibody Complementarity-Determining Regions Bind and Neutralize Human Immunodeficiency Virus Type 1 Particles.

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Biological Characterization of Nef in Long-Term Survivors of Human Immunodeficiency Virus Type 1 Infection.

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A Naturally Occurring Single Basic Amino Acid Substitution in the V3 Region of the Human Immunodeficiency Virus Type 1 Env Protein Alters the Cellular Host Range and Antigenic Structure of the Virus.

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Amanote Research

A Naturally Occurring Variation in the Proline-Rich Region Does Not Attenuate Human Immunodeficiency Virus Type 1 Nef Function

Journal of Virology - United States

doi 10.1128/jvi.78.18.10197-10201.2004

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Human Immunodeficiency Virus Type 1 Nef Does Not Alter T-Cell Sensitivity to Antigen-Specific Stimulation.

Nef-Induced CD4 Downregulation: A Diacidic Sequence in Human Immunodeficiency Virus Type 1 Nef Does Not Function as a Protein Sorting Motif Through Direct Binding to -COP

Dynamic Evolution of the Human Immunodeficiency Virus Type 1 Pathogenic Factor, Nef

Human Immunodeficiency Virus Type 1 Nef: Adapting to Intracellular Trafficking Pathways

Predicting the Impact of Blocking Human Immunodeficiency Virus Type 1 Nef in Vivo

Human Immunodeficiency Virus Type 1 Nef Induces Accumulation of CD4 in Early Endosomes.

Proline-Rich Tandem Repeats of Antibody Complementarity-Determining Regions Bind and Neutralize Human Immunodeficiency Virus Type 1 Particles.

Biological Characterization of Nef in Long-Term Survivors of Human Immunodeficiency Virus Type 1 Infection.

A Naturally Occurring Single Basic Amino Acid Substitution in the V3 Region of the Human Immunodeficiency Virus Type 1 Env Protein Alters the Cellular Host Range and Antigenic Structure of the Virus.