S-Nitrosylation of Β-Arrestin Regulates Β-Adrenergic Receptor Trafficking

Molecular Cell - United States
doi 10.1016/j.molcel.2008.05.024
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Elsevier BV

Related search

Β-Arrestin–biased Signaling Through the Β2-Adrenergic Receptor Promotes Cardiomyocyte Contraction

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
2016English

Mechanism of Β-Adrenergic Receptor Desensitization in Cardiac Hypertrophy Is Increased Β-Adrenergic Receptor Kinase

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
1997English

Phosphorylation Regulates TRPV1 Association With Β-Arrestin-2

Biochemical Journal
BiochemistryCell BiologyMolecular Biology
2013English

Trafficking of Proteinase-Activated Receptor-2 and Β-Arrestin-1 Tagged With Green Fluorescent Protein

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
1999English

Role of Β-Arrestins and Arrestin Domain-Containing Proteins in G Protein-Coupled Receptor Trafficking

Current Opinion in Cell Biology
Cell Biology
2014English

Small Ubiquitin-Like Modifier Modification of Arrestin-3 Regulates Receptor Trafficking

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2010English

Β-Adrenergic Receptor Polymorphisms and Response to Salmeterol

American Journal of Respiratory and Critical Care Medicine
PulmonaryCritical CareRespiratory MedicineIntensive Care Medicine
2006English

Β-Arrestin2 Mediates Β-2 Adrenergic Receptor Signaling Inducing Prostate Cancer Cell Progression

Oncology Reports
MedicineCancer ResearchOncology
2011English

Internalization Determinants of the Parathyroid Hormone Receptor Differentially Regulate Β-Arrestin/Receptor Association

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2001English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy