3-Methylcholanthrene, an AhR Agonist, Caused Cell-Cycle Arrest by Histone Deacetylation Through a RhoA-Dependent Recruitment of HDAC1 and pRb2 to E2F1 Complex

PLoS ONE - United States
doi 10.1371/journal.pone.0092793
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Public Library of Science (PLoS)

Related search

TIGAR Induces P53-Mediated Cell-Cycle Arrest by Regulation of RB–E2F1 Complex

British Journal of Cancer
Cancer ResearchOncology
2012English

The Natural Compound Neobractatin Induces Cell Cycle Arrest by Regulating E2F1 and Gadd45α

Frontiers in Oncology
Cancer ResearchOncology
2019English

Cell Cycle Arrest by Monochloramine Through the Oxidation of Retinoblastoma Protein

Free Radical Biology and Medicine
BiochemistryPhysiology
2003English

Pax7 Activates Myogenic Genes by Recruitment of a Histone Methyltransferase Complex

Nature Cell Biology
Cell Biology
2007English

Histone Deacetylation: IP4 Is an Epigenetic Coregulator

Nature Chemical Biology
Cell BiologyMolecular Biology
2012English

Orostachys Japonicus Induce P53-Dependent Cell Cycle Arrest Through the MAPK Signaling Pathway in OVCAR-3 Human Ovarian Cancer Cells

Food Science and Nutrition
Food Science
2018English

Inhibition of Prostate Cancer Cells by 4,5-Dicaffeoylquinic Acid Through Cell Cycle Arrest

Prostate Cancer
Cancer ResearchOncologyUrology
2019English

Vascular Histone Deacetylation by Pharmacological HDAC Inhibition

Genome Research
Genetics
2014English

Methylation-Mediated Proviral Silencing Is Associated With MeCP2 Recruitment and Localized Histone H3 Deacetylation

Molecular and Cellular Biology
Cell BiologyMolecular Biology
2001English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy