Intramitochondrial Serine Protease Activity of Omi/HtrA2 Is Required for Caspase-Independent Cell Death of Human Neutrophils

Cell Death and Differentiation - United Kingdom
doi 10.1038/sj.cdd.4401409
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Springer Science and Business Media LLC

Related search

Autocatalytic Processing of HtrA2/Omi Is Essential for Induction of Caspase-Dependent Cell Death Through Antagonizing XIAP

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2004English

Binding Specificity and Regulation of the Serine Protease and PDZ Domains of HtrA2/Omi

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2003English

Omi/HtrA2 Protease Associated Cell Apoptosis Participates in Blood-Brain Barrier Dysfunction

Frontiers in Molecular Neuroscience
Molecular NeuroscienceMolecular BiologyCellular
2019English

Rhinovirus 3C Protease Suppresses Apoptosis and Triggers Caspase-Independent Cell Death

Cell Death and Disease
Molecular NeuroscienceImmunologyCell BiologyCancer ResearchCellularMedicine
2018English

Characterization of a Novel and Specific Inhibitor for the Pro-Apoptotic Protease Omi/HtrA2

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2003English

Activation of P38 Mitogen-Activated Protein Kinase Is Required for Death Receptor-Independent Caspase-8 Activation and Cell Death in Response to Sphingosine

Molecular Cancer Research
Cancer ResearchOncologyMolecular Biology
2009English

BCL-2 Prevents Caspase-Independent Cell Death

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
1998English

Blocking Caspase-Independent Death

Journal of Cell Biology
MedicineCell Biology
2002English

Caspase-Independent Programmed Cell Death With Necrotic Morphology

Cell Death and Differentiation
Cell BiologyMolecular Biology
1999English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy