Radiosensitization of Normoxic and Hypoxic H1339 Lung Tumor Cells by Heat Shock Protein 90 Inhibition Is Independent of Hypoxia Inducible Factor-1α

PLoS ONE- United States

doi 10.1371/journal.pone.0031110

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February 7, 2012

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Public Library of Science (PLoS)

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Radiosensitization of Normoxic and Hypoxic H1339 Lung Tumor Cells by Heat Shock Protein 90 Inhibition Is Independent of Hypoxia Inducible Factor-1α

PLoS ONE- United States

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Hypoxia-Inducible Factor 1α (HIF-1α) Protein Is Rapidly Degraded by the Ubiquitin-Proteasome System Under Normoxic Conditions

Inhibition of Protein Synthesis by Imexon Reduces HIF-1α Expression in Normoxic and Hypoxic Pancreatic Cancer Cells

Targeted Replacement of Hypoxia-Inducible Factor-1α by a Hypoxia-Inducible Factor-2α Knock-In Allele Promotes Tumor Growth

Amplification of Tumor Hypoxic Responses by Macrophage Migration Inhibitory Factor-Dependent Hypoxia-Inducible Factor Stabilization

The Tyrosine Phosphatase SHP-1 Regulates Hypoxia Inducible Factor-1α (HIF-1α) Protein Levels in Endothelial Cells Under Hypoxia

Hypoxia-Inducible Factor 1α (HIF1A; HIF1α)

Effects of Hypoxia Inducible Factor-1α on Apoptotic Inhibition and Glucocorticoid Receptor Downregulation by Dexamethasone in AtT-20 Cells

Cooperation of Actin-Sequestering Protein, Thymosin Β-4 and Hypoxia Inducible Factor-1α in Tumor Cell Migration

Identification of Novel Small Molecule Inhibitors of Hypoxia-Inducible Factor-1 That Differentially Block Hypoxia-Inducible Factor-1 Activity and Hypoxia-Inducible Factor-1α Induction in Response to Hypoxic Stress and Growth Factors