(PDF) Organ-Specific Roles for Transcription Factor NF-κB

Organ-Specific Roles for Transcription Factor NF-κB in Reovirus-Induced Apoptosis and Disease

Journal of Clinical Investigation - United States

doi 10.1172/jci22428

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September 1, 2005

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American Society for Clinical Investigation

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Organ-Specific Roles for Transcription Factor NF-κB in Reovirus-Induced Apoptosis and Disease

Journal of Clinical Investigation - United States

doi 10.1172/jci22428

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Tumor Necrosis Factor-Α-Induced Cell Killing and Activation of Transcription Factor NF-κB Are Uncoupled in L929 Cells

Retraction: Modification of Cysteine Residue in P65 Subunit of Nuclear Factor-κB (NF-κB) by Picroliv Suppresses NF-κB–Regulated Gene Products and Potentiates Apoptosis

Apoptosis and NF-κB: The FADD Connection

Apoptosis and NF-κB: The FADD Connection

Hypoxia Induced NF-κB

Modulation of Gene Expression Regulated by the Transcription Factor NF-κB/RelA

Distinct Roles of the IκB Kinase Α and Β Subunits in Liberating Nuclear Factor κB (NF-κB) From IκB and in Phosphorylating the P65 Subunit of NF-κB

NF-κB Mediates Tumor Necrosis Factor Α-Induced Expression of Optineurin, a Negative Regulator of NF-κB

Protein Tyrosine Kinase P56lckIs Required for Ceramide-Induced but Not Tumor Necrosis Factor-Induced Activation of NF-κB, AP-1, JNK, and Apoptosis