Amanote Research
Register
Sign In
Retraction: “Inactivation of Ink4a/Arf Leads to Deregulated Expression of miRNAs in K-Ras Transgenic Mouse Model of Pancreatic Cancer” by Ali Et Al.
Journal of Cellular Physiology
- United States
doi 10.1002/jcp.25416
Full Text
Open PDF
Abstract
Available in
full text
Categories
Clinical Biochemistry
Cell Biology
Physiology
Date
June 17, 2016
Authors
Unknown
Publisher
Wiley
Related search
Retraction: Activated K-Ras and INK4a/Arf Deficiency Cooperate During the Development of Pancreatic Cancer by Activation of Notch and NF-κB Signaling Pathways
PLoS ONE
Multidisciplinary
Activated K-Ras and INK4a/Arf Deficiency Promote Aggressiveness of Pancreatic Cancer by Induction of EMT Consistent With Cancer Stem Cell Phenotype
Journal of Cellular Physiology
Clinical Biochemistry
Cell Biology
Physiology
Retraction: “Concurrent Inhibition of NF-κB, Cyclooxygenase-2, and Epidermal Growth Factor Receptor Leads to Greater Anti-Tumor Activity in Pancreatic Cancer” by Ali Et Al.
Journal of Cellular Biochemistry
Biochemistry
Cell Biology
Molecular Biology
Histone Demethylase JMJD3 Contributes to Epigenetic Control of INK4a/ARF by Oncogenic RAS
Genes and Development
Genetics
Developmental Biology
Ink4a/Arf Expression Is a Biomarker of Aging
Journal of Clinical Investigation
Medicine
A Mouse Model for Tumor Progression of Lung Cancer in Ras and P53 Transgenic Mice
Oncogene
Cancer Research
Genetics
Molecular Biology
MMTV/LTR Promoter-Driven Transgenic Expression of EpCAM Leads to the Development of Large Pancreatic Islets
Journal of Histochemistry and Cytochemistry
Anatomy
Histology
Prognostic Significance of K-Ras Mutations in Pancreatic Cancer: A Meta-Analysis
World Journal of Surgical Oncology
Surgery
Oncology
In Vivo Monitoring of Pancreatic Β-Cells in a Transgenic Mouse Model
Molecular Imaging
Nuclear Medicine
Radiology
Molecular Medicine
Condensed Matter Physics
Imaging
Biotechnology
Biomedical Engineering
