The Cacna1h Mutation in the GAERS Model of Absence Epilepsy Enhances T-Type Ca2+ Currents by Altering Calnexin-Dependent Trafficking of Cav3.2 Channels

Scientific Reports - United Kingdom
doi 10.1038/s41598-017-11591-5
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Springer Science and Business Media LLC

Related search

Ca2+ Signaling by T-Type Ca2+ Channels in Neurons

Pflugers Archiv European Journal of Physiology
Clinical BiochemistryPhysiology
2008English

Reversal of Neuropathic Pain in Diabetes by Targeting Glycosylation of Cav3.2 T-Type Calcium Channels

Diabetes
Internal MedicineEndocrinologyMetabolismDiabetes
2013English

Structural Determinants of the High Affinity Extracellular Zinc Binding Site on Cav3.2 T-Type Calcium Channels

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2009English

Stac Proteins Suppress Ca2+-Dependent Inactivation of Neuronal L-Type Ca2+ Channels

Journal of Neuroscience
Neuroscience
2018English

Functional Expression of T-Type Ca2+ Channels in Spinal Motoneurons of the Adult Turtle

PLoS ONE
Multidisciplinary
2014English

T-Type Ca2+channels Make Your Brain Smarter

Channels
MedicineBiochemistryBiophysics
2015English

Cav3.2 T-Type Calcium Channel Is Required for the NFAT-dependent Sox9 Expression in Tracheal Cartilage

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
2014English

Ca2+/Calmodulin Regulates Trafficking of CaV1.2 Ca2+ Channels in Cultured Hippocampal Neurons

Journal of Neuroscience
Neuroscience
2007English

Selective Increase in T-Type Calcium Conductance of Reticular Thalamic Neurons in a Rat Model of Absence Epilepsy

Journal of Neuroscience
Neuroscience
1995English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy