Blockade of the Tumor Necrosis Factor-Related Apoptosis Inducing Ligand Death Receptor DR5 Prevents Β-Amyloid Neurotoxicity

Neuropsychopharmacology- United Kingdom

doi 10.1038/sj.npp.1301185

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Abstract

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Date

August 16, 2006

Authors

Daniela Uberti

Giulia Ferrari-Toninelli

Publisher

Springer Science and Business Media LLC

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Amanote Research

Blockade of the Tumor Necrosis Factor-Related Apoptosis Inducing Ligand Death Receptor DR5 Prevents Β-Amyloid Neurotoxicity

Neuropsychopharmacology- United Kingdom

Full Text

Abstract

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Date

Authors

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Related search

Inducible Resistance of Tumor Cells to Tumor Necrosis Factor–Related Apoptosis-Inducing Ligand Receptor 2–Mediated Apoptosis by Generation of a Blockade at the Death Domain Function

Tumor Necrosis Factor–Related Apoptosis-Inducing Ligand–Mediated Proliferation of Tumor Cells With Receptor-Proximal Apoptosis Defects

Apo2l/Tumor Necrosis Factor–Related Apoptosis-Inducing Ligand Prevents Breast Cancer–Induced Bone Destruction in a Mouse Model

cFLIPLInhibits Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand-Mediated NF-κB Activation at the Death-Inducing Signaling Complex in Human Keratinocytes

Investigating the Interaction Between Osteoprotegerin and Receptor Activator of NF-κB or Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand

Functional Expression of Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand in Human Colonic Adenocarcinoma Cells

Interferon-Α-Induced Apoptosis via Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand (TRAIL)-dependent and -Independent Manner

Halocynthiaxanthin and Peridinin Sensitize Colon Cancer Cell Lines to Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand

The Oncogene Metadherin Modulates the Apoptotic Pathway Based on the Tumor Necrosis Factor Superfamily Member TRAIL (Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand) in Breast Cancer