Angiotensin-(1-7) Receptor Mas in Hemodynamic and Thermoregulatory Dysfunction After High-Level Spinal Cord Injury in Mice: A Pilot Study

Frontiers in Physiology - Switzerland
doi 10.3389/fphys.2018.01930
Full Text
Abstract

Available in full text

Categories
Physiology
Date
Authors
Publisher

Frontiers Media SA

Related search

Angiotensin 1-7 Protects Against Angiotensin II-Induced Endoplasmic Reticulum Stress and Endothelial Dysfunction via Mas Receptor

PLoS ONE
Multidisciplinary
2015English

Autonomic Dysfunction in Spinal Cord Injury

Juntendo Medical Journal
2016English

Angiotensin-(1-7)-Induced Mas Receptor Activation Attenuates Atherosclerosis Through a Nitric Oxide-Dependent Mechanism in apolipoproteinE-KO Mice

Pflugers Archiv European Journal of Physiology
Clinical BiochemistryPhysiology
2018English

Human Immune Cells Infiltrate the Spinal Cord and Impair Recovery After Spinal Cord Injury in Humanized Mice

Scientific Reports
Multidisciplinary
2019English

Sports, Recreation and Employment Following Spinal Cord Injury–a Pilot Study

Spinal Cord
MedicineNeurologyRehabilitation
2000English

Improved Functional Outcome After Spinal Cord Injury in iNOS-deficient Mice

Spinal Cord
MedicineNeurologyRehabilitation
2004English

Hemodynamic Responses to Head-Up Tilt After Spinal Cord Injury Support a Role for the Mid-Thoracic Spinal Cord in Cardiovascular Regulation

Spinal Cord
MedicineNeurologyRehabilitation
2010English

A Prospective Evaluation of Hemodynamic Management in Acute Spinal Cord Injury Patients

Spinal Cord
MedicineNeurologyRehabilitation
2013English

Interleukin-25 Is Detrimental for Recovery After Spinal Cord Injury in Mice

Journal of Neuroinflammation
Molecular NeuroscienceNeurologyImmunologyCellularNeuroscience
2016English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy