(PDF) Influx in Vivo of Low Density, Intermediate Density,

Influx in Vivo of Low Density, Intermediate Density, and Very Low Density Lipoproteins Into Aortic Intimas of Genetically Hyperlipidemic Rabbits. Roles of Plasma Concentrations, Extent of Aortic Lesion, and Lipoprotein Particle Size as Determinants

Arteriosclerosis and Thrombosis: A Journal of Vascular Biology

doi 10.1161/01.atv.12.1.6

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Date

January 1, 1992

Authors

B G Nordestgaard

A Tybjaerg-Hansen

B Lewis

Publisher

Ovid Technologies (Wolters Kluwer Health)

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Influx in Vivo of Low Density, Intermediate Density, and Very Low Density Lipoproteins Into Aortic Intimas of Genetically Hyperlipidemic Rabbits. Roles of Plasma Concentrations, Extent of Aortic Lesion, and Lipoprotein Particle Size as Determinants

Arteriosclerosis and Thrombosis: A Journal of Vascular Biology

doi 10.1161/01.atv.12.1.6

Full Text

Abstract

Date

Authors

Publisher

Related search

Development of an Integrated Model for Analysis of the Kinetics of Apolipoprotein B in Plasma Very Low Density Lipoproteins, Intermediate Density Lipoproteins, and Low Density Lipoproteins.

Effects of Very Low Density Lipoprotein From Watanabe Heritable Hyperlipidemic Rabbits on Foam Cell Formation

The Low-Density-Lipoprotein Pathway of Native and Chemically Modified Low-Density Lipoproteins Isolated From Plasma Incubatedin Vitro

Effects of Low-Dose Oral Contraceptives on Very Low-Density and Low-Density Lipoprotein Metabolism

Neonatal Umbilical Cord Blood Lipoproteins. Isolation and Characterization of Intermediate Density and Low Density Lipoproteins

Metabolism of Very Low Density Lipoproteins in Genetically Lean or Fat Lines of Chicken

Association of Angiotensin-Converting Enzyme With Low-Density Lipoprotein in Aortic Valvular Lesions and in Human Plasma

Overexpression of Hepatic Lipase in Transgenic Rabbits Leads to a Marked Reduction of Plasma High Density Lipoproteins and Intermediate Density Lipoproteins.

Pre-Beta-Very Low Density Lipoproteins as Precursors of Beta-Very Low Density Lipoproteins. A Model for the Pathogenesis of Familial Dysbetalipoproteinemia (Type III Hyperlipoproteinemia).