Post-Ischemic Early Acidosis in Cardiac Postconditioning Modifies the Activity of Antioxidant Enzymes, Reduces Nitration, and Favors Protein S-Nitrosylation

Pflugers Archiv European Journal of Physiology - Germany
doi 10.1007/s00424-011-0970-1
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Springer Science and Business Media LLC

Related search

NOS1, S-Nitrosylation and Cardiac Contraction

2011English

S-Nitrosylation of Cardiac Ion Channels

Journal of Cardiovascular Pharmacology
Cardiovascular MedicinePharmacologyCardiology
2009English

Nitric Oxide Reduces NADPH Oxidase 5 (Nox5) Activity by Reversible S-Nitrosylation

Free Radical Biology and Medicine
BiochemistryPhysiology
2012English

Radial Growth and Activity of Antioxidant Enzymes in the Three Post-Radiation Cladosporium Cladosporioides Generations

Nuclear Physics and Atomic Energy
High Energy PhysicsNuclear
2017English

Mitochondria in Cardiac Postconditioning

Frontiers in Physiology
Physiology
2018English

Acidic Infusion in Early Reperfusion Affects the Activity of Antioxidant Enzymes in Postischemic Isolated Rat Heart

Journal of Surgical Research
Surgery
2013English

Identification of S-Nitroso-CoA Reductases That Regulate Protein S-Nitrosylation

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
2014English

dATP Reduces the Depressive Effect of Acidosis on Cardiac and Slow Twitch Skeletal Muscle

Biophysical Journal
Biophysics
2019English

Detection of Protein S-Nitrosylation With the Biotin-Switch Technique

Free Radical Biology and Medicine
BiochemistryPhysiology
2009English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy