ER Stress-Induced eIF2-alpha Phosphorylation Underlies Sensitivity of Striatal Neurons to Pathogenic Huntingtin

PLoS ONE - United States
doi 10.1371/journal.pone.0090803
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Public Library of Science (PLoS)

Related search

Behavioral Sensitivity of Temporally Modulated Striatal Neurons

Frontiers in Integrative Neuroscience
Molecular NeuroscienceSensory SystemsCellularCognitive Neuroscience
2011English

Icariin Induces Synoviolin Expression Through NFE2L1 to Protect Neurons From ER Stress-Induced Apoptosis

PLoS ONE
Multidisciplinary
2015English

UPR, Autophagy, and Mitochondria Crosstalk Underlies the ER Stress Response

Trends in Biochemical Sciences
BiochemistryMolecular Biology
2015English

Mitogen- And Stress-Activated Protein Kinase-1 Deficiency Is Involved in Expanded-Huntingtin-Induced Transcriptional Dysregulation and Striatal Death

FASEB Journal
BiochemistryBiotechnologyGeneticsMolecular BiologyMedicine
2007English

Reduced Slc6a15 in Nucleus Accumbens D2-Neurons Underlies Stress Susceptibility

Journal of Neuroscience
Neuroscience
2017English

Stress Induced Alterations in Striatal GABAA Receptor Complex.

The Japanese Journal of Pharmacology
1990English

Metabotropic Glutamate Receptors and Dopamine Receptors Cooperate to Enhance Extracellular Signal-Regulated Kinase Phosphorylation in Striatal Neurons

Journal of Neuroscience
Neuroscience
2005English

Current Concepts in ER Stress-Induced Apoptosis

Journal of Carcinogenesis & Mutagenesis
2013English

Elongation Factor-1 Alpha Is a Selective Regulator of Growth Factor Withdrawal and ER Stress-Induced Apoptosis

Cell Death and Differentiation
Cell BiologyMolecular Biology
2002English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy