Decreased Striatal Dopamine Release Underlies Increased Expression of Long-Term Synaptic Potentiation at Corticostriatal Synapses 24 H After 3-Nitropropionic-Acid-Induced Chemical Hypoxia

Journal of Neuroscience - United States
doi 10.1523/jneurosci.5698-07.2008
Full Text
Abstract

Available in full text

Categories
Neuroscience
Date
Authors
Publisher

Society for Neuroscience

Related search

Remodeling of Synaptic Membranes After Induction of Long-Term Potentiation

Journal of Neuroscience
Neuroscience
2001English

Noncanonical, Dopamine-Dependent Long-Term Potentiation at Hippocampal Output Synapses in a Rodent Model of First-Episode Psychosis

Frontiers in Molecular Neuroscience
Molecular NeuroscienceMolecular BiologyCellular
2020English

Phorbol Ester-Induced Synaptic Facilitation Is Different Than Long-Term Potentiation.

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
1988English

Nicotinic Acetylcholine Receptors at Glutamate Synapses Facilitate Long-Term Depression or Potentiation

Journal of Neuroscience
Neuroscience
2005English

SNAP-25 Isoforms Differentially Regulate Synaptic Transmission and Long-Term Synaptic Plasticity at Central Synapses

Scientific Reports
Multidisciplinary
2019English

Dopamine Modulates Release From Corticostriatal Terminals

Journal of Neuroscience
Neuroscience
2004English

SCRAPPER Regulates the Thresholds of Long-Term Potentiation/Depression, the Bidirectional Synaptic Plasticity in Hippocampal CA3-CA1 Synapses

Neural Plasticity
Neurology
2012English

Methamphetamine-Induced Vascular Changes Lead to Striatal Hypoxia and Dopamine Reduction

NeuroReport
Neuroscience
2011English

Orphenadrine Prevents 3-Nitropropionic Acid-Induced Neurotoxicityin Vitroandin Vivo

British Journal of Pharmacology
Pharmacology
2001English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy