Paradoxical Activation of T Cells via Augmented ERK Signaling Mediated by a RAF Inhibitor

Cancer immunology research- United States

doi 10.1158/2326-6066.cir-13-0160

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October 14, 2013

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American Association for Cancer Research (AACR)

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Paradoxical Activation of T Cells via Augmented ERK Signaling Mediated by a RAF Inhibitor

Cancer immunology research- United States

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Time-Resolved Phosphoproteome Analysis of Paradoxical RAF Activation Reveals Novel Targets of ERK

Combination of a Selective HSP90α/β Inhibitor and a RAS-RAF-MEK-ERK Signaling Pathway Inhibitor Triggers Synergistic Cytotoxicity in Multiple Myeloma Cells

Activation of CD4 T Cells by Raf-Independent Effectors of Ras

Next-Generation RAF Inhibitors Prevent Paradoxical MAPK Activation

930 SOX2 Epidermal Overexpression Promotes Cutaneous Wound Healing via Activation of EGFR/MEK/ERK Signaling Mediated by EGFR Ligands

EIF5A1 Promotes Trophoblast Migration and Invasion via ARAF-mediated Activation of the Integrin/Erk Signaling Pathway

Molecular Signaling in Multiple Myeloma: Association of RAS/RAF Mutations and MEK/ERK Pathway Activation

Neural Stem Cells Secrete Factors Facilitating Brain Regeneration Upon Constitutive Raf-Erk Activation

Receptor Heterodimerization Leads to a Switch in Signaling: -Arrestin2-Mediated ERK Activation by - Opioid Receptor Heterodimers