(PDF) Potential Role for Luman, the Cellular Homologue of

Potential Role for Luman, the Cellular Homologue of Herpes Simplex Virus VP16 (Alpha Gene Trans-Inducing Factor), in Herpesvirus Latency

Journal of Virology - United States

doi 10.1128/jvi.74.2.934-943.2000

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Date

January 15, 2000

Authors

R. Lu

V. Misra

Publisher

American Society for Microbiology

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Potential Role for Luman, the Cellular Homologue of Herpes Simplex Virus VP16 (Alpha Gene Trans-Inducing Factor), in Herpesvirus Latency

Journal of Virology - United States

doi 10.1128/jvi.74.2.934-943.2000

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Herpes Simplex Virus Type 1 and Bovine Herpesvirus 1 Latency

Experimental Investigation of Herpes Simplex Virus Latency.

The Latency-Associated Transcript Gene Enhances Establishment of Herpes Simplex Virus Type 1 Latency in Rabbits

Protein Interactions in the Herpes Simplex Virus Type 1 VP16-induced Complex: VP16 Peptide Inhibition and Mutational Analysis of Host Cell Factor Requirements.

Upstream Stimulatory Factor Family Binds to the Herpes Simplex Virus Type 1 Latency-Associated Transcript Promoter

Antiviral Therapy for Herpesvirus Central Nervous System Infections: Neonatal Herpes Simplex Virus Infection, Herpes Simplex Encephalitis, and Congenital Cytomegalovirus Infection

Nuclear Localization of Herpesvirus Proteins: Potential Role for the Cellular Framework.

Evidence That Two Latency-Associated Transcripts of Herpes Simplex Virus Type 1 Are Nonlinear.

The VP16 Paradox: Herpes Simplex Virus VP16 Contains a Long-Range Activation Domain but Within the Natural Multiprotein Complex Activates Only From Promoter-Proximal Positions.