Amyloid-Β1-42 Disrupts Synaptic Plasticity by Altering Glutamate Recycling at the Synapse

Journal of Alzheimer's Disease - Netherlands
doi 10.3233/jad-142367
Full Text
Abstract

Available in full text

Date
Authors
Publisher

IOS Press

Related search

Okadaic Acid Disrupts Synaptic Vesicle Trafficking in a Ribbon-Type Synapse

Journal of Neurochemistry
BiochemistryMolecular NeuroscienceCellular
2004English

BARS at the Synapse: PICK-1 Lipid Binding Domain Regulates Targeting, Trafficking, and Synaptic Plasticity

Journal of Neuroscience
Neuroscience
2006English

Molecular Signals of Plasticity at the Tetrapartite Synapse

Current Opinion in Neurobiology
Neuroscience
2011English

Defective Synapse Maturation and Enhanced Synaptic Plasticity in Shank2 Δex7 –/– Mice

eNeuro
MedicineNeuroscience
2018English

Analysis of TCR/CD3 Recycling at the Immune Synapse

Methods in Molecular Biology
GeneticsMolecular Biology
2017English

Quantitative Modeling of Synaptic Release at the Photoreceptor Synapse

Biophysical Journal
Biophysics
2010English

Emerging Treatment Mechanisms for Depression: Focus on Glutamate and Synaptic Plasticity

Drug Discovery Today
Drug DiscoveryPharmacology
2016English

Synaptic Plasticity Disruption by Amyloid Β Protein: Modulation by Potential Alzheimer's Disease Modifying Therapies: Figure 1

Biochemical Society Transactions
Biochemistry
2005English

Quantitative Analysis of Vesicle Recycling at the Calyx of Held Synapse

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
2015English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy