A Mouse Model of Human Congenital Heart Disease: High Incidence of Diverse Cardiac Anomalies and Ventricular Noncompaction Produced by Heterozygous Nkx2-5 Homeodomain Missense Mutation

Circulation: Cardiovascular Genetics
doi 10.1161/circgenetics.113.000281
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Ovid Technologies (Wolters Kluwer Health)

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