Mutations in Caenorhabditis Elegans Actin, Which Are Equivalent to Human Cardiomyopathy Mutations, Cause Abnormal Actin Aggregation in Nematode Striated Muscle

F1000Research- United Kingdom

doi 10.12688/f1000research.18476.1

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March 12, 2019

Authors

Yuriko Hayashi

Kanako Ono

Shoichiro Ono

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F1000 Research Ltd

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Mutations in Caenorhabditis Elegans Actin, Which Are Equivalent to Human Cardiomyopathy Mutations, Cause Abnormal Actin Aggregation in Nematode Striated Muscle

F1000Research- United Kingdom

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Developmental Genetic Analysis of Troponin T Mutations in Striated and Nonstriated Muscle Cells of Caenorhabditis Elegans

Clinical and Genetic Characteristics of Cardiac Actin Gene Mutations in Hypertrophic Cardiomyopathy

Effects of Human Deafness Γ-Actin Mutations (DFNA20/26) on Actin Function

Corrigendum: Mutations in Smooth Muscle Α-Actin (ACTA2) Lead to Thoracic Aortic Aneurysms and Dissections

Tropomodulin Capping of Actin Filaments in Striated Muscle Development and Physiology

Myo1c Mutations Associated With Hearing Loss Cause Defects in the Interaction With Nucleotide and Actin

Mutations in Possible Glycosylation Genes, SQV-3,7 and 8, of C. Elegans Cause Abnormal Vulval Formation.

Microsporidia Are Natural Intracellular Parasites of the Nematode Caenorhabditis Elegans

Modulation of Striated Muscle Contraction by Binding of Myosin Binding Protein C to Actin