(PDF) Activation of Rac GTPase by P75 Is Necessary for

Activation of Rac GTPase by P75 Is Necessary for C-junN-Terminal Kinase-Mediated Apoptosis

Journal of Neuroscience - United States

doi 10.1523/jneurosci.22-01-00156.2002

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Date

January 1, 2002

Authors

Anthony W. Harrington

Ju Young Kim

Sung Ok Yoon

Publisher

Society for Neuroscience

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Amanote Research

Activation of Rac GTPase by P75 Is Necessary for C-junN-Terminal Kinase-Mediated Apoptosis

Journal of Neuroscience - United States

doi 10.1523/jneurosci.22-01-00156.2002

Full Text

Abstract

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Date

Authors

Publisher

Related search

GqProtein-induced Apoptosis Is Mediated by AKT Kinase Inhibition That Leads to Protein Kinase C-Induced C-Jun N-Terminal Kinase Activation

Activation of C-Jun-N-Terminal Kinase Is Required for Apoptosis Triggered by Glutathione Disulfide in Neuroblastoma Cells

C-Jun NH2-Terminal Kinase-Mediated Signaling Is Essential for Pseudomonas Aeruginosa ExoS-Induced Apoptosis

The C-Terminal Activation Domain of the STAT-1 Transcription Factor Is Necessary and Sufficient for Stress-Induced Apoptosis

JNK (C-Jun N-Terminal Kinase) and P38 Activation in Receptor-Mediated and Chemically-Induced Apoptosis of T-Cells: Differential Requirements for Caspase Activation

Prolactin-Releasing Peptide Activation of the Prolactin Promoter Is Differentially Mediated by Extracellular Signal-Regulated Protein Kinase and C-Jun N-Terminal Protein Kinase

Rac GTPase Interacts Specifically With Phosphatidylinositol 3-Kinase

Spontaneous Neutrophil Apoptosis Involves Caspase 3-Mediated Activation of Protein Kinase C-Δ

Activation of Stress-Activated Protein Kinase/C-Jun NH2-terminal Kinase and P38 Kinase in Calphostin C-Induced Apoptosis Requires Caspase-3-Like Proteases but Is Dispensable for Cell Death