Prevention of Burn-Induced Inflammatory Responses and Muscle Wasting by GTS-21, a Specific Agonist for Α7 Nicotinic Acetylcholine Receptors

Shock - United States
doi 10.1097/shk.0000000000000729
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Ovid Technologies (Wolters Kluwer Health)

Related search

Two Neuronal Nicotinic Acetylcholine Receptors, Α4β4 and Α7, Show Differential Agonist Binding Modes.

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2011English

Α7 Nicotinic Acetylcholine Receptors in Lung Cancer (Review)

Oncology Letters
Cancer ResearchOncology
2018English

Nicotinic Acetylcholine Receptor Α7 (CHRNA7)

Science-Business eXchange
2010English

Nicotinic Acetylcholine Receptor Α7 (CHRNA7)

Science-Business eXchange
2010English

Nicotinic Acetylcholine Receptor Α7 (CHRNA7)

Science-Business eXchange
2013English

Sulfonium as a Surrogate for Ammonium: A New Α7 Nicotinic Acetylcholine Receptor Partial Agonist With Desensitizing Activity.

Journal of Medicinal Chemistry
Drug DiscoveryMolecular Medicine
2017English

Effects of the Nicotinic Α7 Receptor Partial Agonist GTS-21 on NMDA-glutamatergic Receptor Related Deficits in Sensorimotor Gating and Recognition Memory in Rats

Psychopharmacology
Pharmacology
2014English

Identification and in Vitro Pharmacological Characterization of a Novel and Selective Α7 Nicotinic Acetylcholine Receptor Agonist, Br-Iq17b

Acta Pharmacologica Sinica
MedicinePharmacology
2015English

Asymmetric Agonist Selectivity of Agonist Sites in (Α4β2)2α4 Nicotinic Acetylcholine Receptors: A Key Determinant of Agonist Efficacy

Biophysical Journal
Biophysics
2014English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy