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Differential Sensitivity of Endothelial Cells of Various Species to Apoptosis Induced by Gene Transfer of Fas Ligand: Role of Flip Levels

Molecular Medicine - United States

doi 10.1007/bf03402172

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October 1, 2002

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Springer Science and Business Media LLC

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Differential Sensitivity of Endothelial Cells of Various Species to Apoptosis Induced by Gene Transfer of Fas Ligand: Role of Flip Levels

Molecular Medicine - United States

doi 10.1007/bf03402172

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

CK2 Controls TRAIL and Fas Sensitivity by Regulating FLIP Levels in Endometrial Carcinoma Cells

Role of P38 Mitogen-Activated Protein Kinase Phosphorylation and Fas-Fas Ligand Interaction in Morphine-Induced Macrophage Apoptosis

Involvement of Gene Expressions in Apoptosis of Vascular Endothelial Cells Induced by Rattlesnake Venom

Adenovirus-Mediated Transfer of P53 and Fas Ligand Drastically Enhances Apoptosis in Gliomas

Inhibition of Fas-Induced Apoptosis by BCL-2

Interleukin-12 Enhances the Sensitivity of Human Osteosarcoma Cells to 4-Hydroperoxycyclophosphamide by a Mechanism Involving the Fas/Fas-Ligand Pathway

Involvement of Fas (CD95/APO-1) and Fas Ligand in Apoptosis Induced by Ganciclovir Treatment of Tumor Cells Transduced With Herpes Simplex Virus Thymidine Kinase

Hypoxia Stimulates Release of the Soluble Form of Fas Ligand That Inhibits Endothelial Cell Apoptosis

Levels of Glucocorticoid Receptor and Its Ligand Determine Sensitivity and Kinetics of Glucocorticoid-Induced Leukemia Apoptosis