Transcription Factor YY1 Contributes to Tumor Growth by Stabilizing Hypoxia Factor HIF-1 in a P53-Independent Manner

Cancer Research- United States

doi 10.1158/0008-5472.can-12-0366

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January 17, 2013

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American Association for Cancer Research (AACR)

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Transcription Factor YY1 Contributes to Tumor Growth by Stabilizing Hypoxia Factor HIF-1 in a P53-Independent Manner

Cancer Research- United States

Full Text

Abstract

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Date

Authors

Publisher

Related search

P300 Relieves P53-Evoked Transcriptional Repression of Hypoxia-Inducible Factor-1 (HIF-1)

Transcriptional Regulatory Mechanism by the Transcription Factor, Hypoxia-Inducible Factor 1, Activated in Hypoxia.

Tissue Expression of Hypoxia-Inducible Factor 1 Alpha (HIF-1A) in Wilms Tumor

Small-Molecule Activation of P53 Blocks Hypoxia-Inducible Factor 1 and Vascular Endothelial Growth Factor Expression in Vivo and Leads to Tumor Cell Apoptosis in Normoxia and Hypoxia

Enhanced Hypoxia-Inducible Factor (HIF)-1α Stability Induced by 5-Hydroxymethyl-2-Furfural (5-Hmf) Contributes to Protection Against Hypoxia

Growth Factor Independent 1 Is a Tumor Suppressor Gene in Colorectal Cancer

A Dynamic Model of the Hypoxia-Inducible Factor 1 (HIF-1 ) Network

Targeted Replacement of Hypoxia-Inducible Factor-1α by a Hypoxia-Inducible Factor-2α Knock-In Allele Promotes Tumor Growth

Transcription Factor ATF2 Cooperates With V-Jun to Promote Growth Factor-Independent Proliferation in Vitro and Tumor Formation in Vivo