Complement in Experimental Autoimmune Encephalomyelitis Revisited: C3 Is Required for Development of Maximal Disease

Molecular Immunology - United Kingdom
doi 10.1016/j.molimm.2007.02.002
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Elsevier BV

Related search

Effects of Complement C5 on Apoptosis in Experimental Autoimmune Encephalomyelitis

Journal of Immunology
AllergyImmunology
2004English

The NF- B Transcription Factor C-Rel Is Required for Th17 Effector Cell Development in Experimental Autoimmune Encephalomyelitis

Journal of Immunology
AllergyImmunology
2011English

The Complement Anaphylatoxin Receptors Are Not Required for the Development of Experimental Autoimmune Uveitis

Journal of Neuroimmunology
AllergyImmunologyNeurology
2013English

Fatal Neurological Disease in Scrapie-Infected Mice Induced for Experimental Autoimmune Encephalomyelitis

Journal of Virology
Insect ScienceImmunologyMicrobiologyVirology
2007English

MAdCAM-1-Mediated Intestinal Lymphocyte Homing Is Critical for the Development of Active Experimental Autoimmune Encephalomyelitis

Frontiers in Immunology
AllergyImmunology
2019English

IL-21R Signaling Is Critical for Induction of Spontaneous Experimental Autoimmune Encephalomyelitis

Journal of Clinical Investigation
Medicine
2015English

Mechanical Hypernociception in Experimental Autoimmune Encephalomyelitis

Arquivos de Neuro-Psiquiatria
Biological PsychiatryNeurology
2009English

Complement Therapeutics in Autoimmune Disease

Frontiers in Immunology
AllergyImmunology
2019English

The Experimental Autoimmune Encephalomyelitis Disease Course Is Modulated by Nicotine and Other Cigarette Smoke Components

PLoS ONE
Multidisciplinary
2014English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy