Tiotropium Bromide Inhibits TGF-ß-induced MMP Production From Lung Fibroblasts by Interfering With Smad and MAPK Pathways in Vitro

International Journal of COPD- New Zealand

doi 10.2147/copd.s11737

Full Text

Abstract

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Date

August 1, 2010

Authors

Kazuhito Asano

Publisher

Informa UK Limited

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Tiotropium Bromide Inhibits TGF-ß-induced MMP Production From Lung Fibroblasts by Interfering With Smad and MAPK Pathways in Vitro

International Journal of COPD- New Zealand

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

TGF-beta1 Stimulates Human AT1 Receptor Expression in Lung Fibroblasts by Cross Talk Between the Smad, P38 MAPK, JNK, and PI3K Signaling Pathways

Rhynchophylline Attenuates Allergic Bronchial Asthma by Inhibiting TGF-1-induced Smad and MAPK Signaling Transductions in Vivo and in Vitro

Fucoxanthin Inhibits Myofibroblast Differentiation and Extracellular Matrix Production in Nasal Polyp-Derived Fibroblasts via Modulation of Smad-Dependent and Smad-Independent Signaling Pathways

Relaxin Inhibits High Glucose-Induced Matrix Accumulation in Human Mesangial Cells by Interfering With TGF-β1 Production and Mesangial Cells Phenotypic Transition

Fluorofenidone Affects Hepatic Stellate Cell Activation in Hepatic Fibrosis by Targeting the TGF‑β1/Smad and MAPK Signaling Pathways

Erythromycin Modulates Eosinophil Chemotactic Cytokine Production by Human Lung Fibroblasts in Vitro

Tiotropium Bromide: An Update

Curcumin Inhibits EMMPRIN and MMP-9 Expression Through AMPK-MAPK and PKC Signaling in PMA Induced Macrophages

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Tiotropium Bromide Inhibits TGF-&szlig;-induced MMP Production From Lung Fibroblasts by Interfering With Smad and MAPK Pathways in Vitro

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TGF-beta1 Stimulates Human AT1 Receptor Expression in Lung Fibroblasts by Cross Talk Between the Smad, P38 MAPK, JNK, and PI3K Signaling Pathways

Rhynchophylline Attenuates Allergic Bronchial Asthma by Inhibiting TGF-1-induced Smad and MAPK Signaling Transductions in Vivo and in Vitro

Fucoxanthin Inhibits Myofibroblast Differentiation and Extracellular Matrix Production in Nasal Polyp-Derived Fibroblasts via Modulation of Smad-Dependent and Smad-Independent Signaling Pathways

Relaxin Inhibits High Glucose-Induced Matrix Accumulation in Human Mesangial Cells by Interfering With TGF-β1 Production and Mesangial Cells Phenotypic Transition

Fluorofenidone Affects Hepatic Stellate Cell Activation in Hepatic Fibrosis by Targeting the TGF‑β1/Smad and MAPK Signaling Pathways

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Tiotropium Bromide Inhibits TGF-ß-induced MMP Production From Lung Fibroblasts by Interfering With Smad and MAPK Pathways in Vitro