Cofilin-Actin Rod Formation in Experimental Stroke Is Attenuated by Therapeutic Hypothermia and Overexpression of the Inducible 70 kD Inducible Heat Shock Protein (Hsp70)

Brain Circulation
doi 10.4103/bc.bc_52_19
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Medknow

Related search

A Small Molecule Inhibitor of Inducible Heat Shock Protein 70

Molecular Cell
Cell BiologyMolecular Biology
2009English

Description of Strongly Heat-Inducible Heat Shock Protein 70 Transcripts From Baikal Endemic Amphipods

Scientific Reports
Multidisciplinary
2019English

Overexpression of Inducible 70-kDa Heat Shock Protein in Mouse Improves Structural and Functional Recovery of Skeletal Muscles From Atrophy

Pflugers Archiv European Journal of Physiology
Clinical BiochemistryPhysiology
2012English

Proteasome and Heat Shock Protein 70 (HSP70) Inhibitors as Therapeutic Alternative in Multiple Myeloma

Oncotarget
Oncology
2017English

cDNA Cloning of Heat-Inducible HSP70, a 70.6 kDa Heat Shock Protein, in Japanese Flounder Paralichtys Olivaceus

Fisheries Science
Aquatic Science
1998English

Anti-Inflammatory Effects of the 70 kDa Heat Shock Protein in Experimental Stroke

Journal of Cerebral Blood Flow and Metabolism
NeurologyCardiovascular MedicineCardiology
2007English

Cofilin Nuclear-Cytoplasmic Shuttling Affects Cofilin-Actin Rod Formation During Stress

Journal of Cell Science
Cell Biology
2012English

Crystal Structure of the Stress-Inducible Human Heat Shock Protein 70 Substrate-Binding Domain in Complex With Peptide Substrate

PLoS ONE
Multidisciplinary
2014English

Expression of the Constitutive and Inducible Forms of Heat Shock Protein 70 in Human Proximal Tubule Cells Exposed to Heat, Sodium Arsenite, and CdCl(2)

Environmental Health Perspectives
MutagenesisPublic HealthEnvironmentalHealthToxicologyOccupational Health
1999English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy