Erythrocytes From Hereditary Xerocytosis Patients Heterozygous for KCNN4 V282M Exhibit Increased Spontaneous Gardos Channel-Like Activity Inhibited by Senicapoc

American Journal of Hematology- United States

doi 10.1002/ajh.24716

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April 29, 2017

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Wiley

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Amanote Research

Erythrocytes From Hereditary Xerocytosis Patients Heterozygous for KCNN4 V282M Exhibit Increased Spontaneous Gardos Channel-Like Activity Inhibited by Senicapoc

American Journal of Hematology- United States

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Red Blood Cell Gardos Channel (KCNN4):the Essential Determinant of Erythrocyte Dehydration in Hereditary Xerocytosis

Hypoxic Exercise Training Causes Erythrocyte Senescence and Rheological Dysfunction by Depressed Gardos Channel Activity

Hemoglobin C Trait Accentuates Erythrocyte Dehydration in Hereditary Xerocytosis

Arginine Supplementation of Sickle Transgenic Mice Reduces Red Cell Density and Gardos Channel Activity

Xerocytosis Is Caused by Mutations That Alter the Kinetics of the Mechanosensitive Channel PIEZO1

Solenopsin a and Analogs Exhibit Ceramide-Like Biological Activity

Abnormal Na-K-ATPase Activity in Erythrocytes From Patients With Diabetes Mellitus

Mechanical Properties of Model Erythrocytes Membranes From Healthy and Hereditary Spherocytosis Subjects

Norepinephrine Transporter Heterozygous Knockout Mice Exhibit Altered Transport and Behavior