By Improving Regional Cortical Blood Flow, Attenuating Mitochondrial Dysfunction and Sequential Apoptosis Galangin Acts as a Potential Neuroprotective Agent After Acute Ischemic Stroke

Molecules - Switzerland
doi 10.3390/molecules171113403
Full Text
Abstract

Available in full text

Date
Authors
Publisher

MDPI AG

Related search

Normobaric Hyperoxygenation: A Potential Neuroprotective Therapy for Acute Ischemic Stroke?

Expert Review of Neurotherapeutics
NeuroscienceNeurologyPharmacology
2017English

Induced Hypertension Improves Cerebral Blood Flow in Acute Ischemic Stroke

Neurology
Neurology
2005English

Nicotine as a Potential Neuroprotective Agent for Parkinson's Disease

Movement Disorders
Neurology
2012English

Blood Pressure Variability and Hemorrhagic Transformation After Intravenous Thrombolysis in Acute Ischemic Stroke

Journal of Neurology and Neuromedicine
2016English

Prediction of Adverse Outcomes by Blood Glucose Level After Endovascular Therapy for Acute Ischemic Stroke

Journal of Neurosurgery
SurgeryNeurology
2011English

Optimal Blood Pressure After Reperfusion Therapy in Patients With Acute Ischemic Stroke

Scientific Reports
Multidisciplinary
2019English

Therapeutic Target and Cell-Signal Communication of Chlorpromazine and Promethazine in Attenuating Blood–Brain Barrier Disruption After Ischemic Stroke

Cell Transplantation
TransplantationBiomedical EngineeringCell Biology
2018English

Subarachnoid Pharmacotherapy for Maximizing Recovery After Cortical Ischemic Stroke

Journal of Experimental Stroke and Translational Medicine
NeuroscienceNeurologyCognitive Neuroscience
2010English

Brain–heart Interaction After Acute Ischemic Stroke

Critical Care
Critical CareIntensive Care Medicine
2020English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy