Cigarette Smoke Condensate and Dioxin Suppress Culture Shock Induced Senescence in Normal Human Oral Keratinocytes

Oral Oncology- United Kingdom

doi 10.1016/j.oraloncology.2006.08.008

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August 1, 2007

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Elsevier BV

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Cigarette Smoke Condensate and Dioxin Suppress Culture Shock Induced Senescence in Normal Human Oral Keratinocytes

Oral Oncology- United Kingdom

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Mechanism of Cigarette Smoke Condensate-Induced Acute Inflammatory Response in Human Bronchial Epithelial Cells

Cigarette Smoke Condensate Increases Cathepsin-Mediated Invasiveness of Oral Carcinoma Cells

Cytotoxicity of HBD3 for Dendritic Cells, Normal Human Epidermal Keratinocytes, hTERT Keratinocytes, and Primary Oral Gingival Epithelial Keratinocytes in Cell Culture Conditions

Fractionation of Mouse Skin Carcinogens in Cigarette Smoke Condensate

14,15-Epoxyeicosatrienoic Acid Suppresses Cigarette Smoke Condensate-Induced Inflammation in Lung Epithelial Cells by Inhibiting Autophagy

AP-2α Downregulation by Cigarette Smoke Condensate Is Counteracted by P53 in Human Lung Cancer Cells

The Direct Preparation of Cigarette Smoke Condensate by High Velocity Impaction

Kinetin-Induced Differentiation of Normal Human Keratinocytes Undergoing Aging in Vitro

Estrogen Receptor Α Increases Basal and Cigarette Smoke Extract-Induced Expression ofCYP1A1 andCYP1B1, but notGSTP1, in Normal Human Bronchial Epithelial Cells