(PDF) Live-Cell Imaging of Endogenous Ras-GTP Illustrates

Live-Cell Imaging of Endogenous Ras-GTP Illustrates Predominant Ras Activation at the Plasma Membrane

EMBO Reports - United States

doi 10.1038/sj.embor.7400560

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January 1, 2006

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Amanote Research

Live-Cell Imaging of Endogenous Ras-GTP Illustrates Predominant Ras Activation at the Plasma Membrane

EMBO Reports - United States

doi 10.1038/sj.embor.7400560

Full Text

Abstract

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Date

Authors

Publisher

Related search

The Structural Basis for the Transition From Ras-GTP to Ras-GDP

The Deubiquitinating Enzyme USP17 Blocks N-Ras Membrane Trafficking and Activation but Leaves K-Ras Unaffected

Galectin-1 Binds Oncogenic H-Ras to Mediate Ras Membrane Anchorage and Cell Transformation

Colocalization of Ras and Ral on the Membrane Is Required for Ras-Dependent Ral Activation Through Ral GDP Dissociation Stimulator

In Vitro Expression of NGN3 Identifies RAB3B as the Predominant Ras-Associated GTP-binding Protein 3 Family Member in Human Islets

Galectin-1 Augments Ras Activation and Diverts Ras Signals to Raf-1 at the Expense of Phosphoinositide 3-Kinase

Ras-GTP Dimers Activate the Mitogen-Activated Protein Kinase (MAPK) Pathway

Raf Activation by Ras and Promotion of Cellular Metastasis Require Phosphorylation of Prohibitin in the Raft Domain of the Plasma Membrane

Differential Interaction of the Ras Family GTP-binding Proteins H-Ras, Rap1A, and R-Ras With the Putative Effector Molecules Raf Kinase and Ral-Guanine Nucleotide Exchange Factor