Trimetazidine Attenuates Cardiac Dysfunction in Endotoxemia and Sepsis by Promoting Neutrophil Migration

Frontiers in Immunology - Switzerland
doi 10.3389/fimmu.2018.02015
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Frontiers Media SA

Related search

MicroRNA-155 Attenuates Late Sepsis-Induced Cardiac Dysfunction Through JNK and Β-Arrestin 2

Oncotarget
Oncology
2017English

Erythropoietin Attenuates Cardiac Dysfunction in Experimental Sepsis in Mice via Activation of the -Common Receptor

DMM Disease Models and Mechanisms
ImmunologyMolecular BiologyBiochemistryMicrobiology NeuroscienceMedicineGenetics
2013English

233 Erythropoietin Attenuates Cardiac Dysfunction in Experimental Sepsis via Activation of the Β-Common Receptor

Heart
Cardiovascular MedicineCardiology
2013English

Rac1 Activation Induces Tumour Necrosis Factor-Α Expression and Cardiac Dysfunction in Endotoxemia

Journal of Cellular and Molecular Medicine
Molecular MedicineCell Biology
2010English

Simvastatin Attenuates Oxidant-Induced Mitochondrial Dysfunction in Cardiac Myocytes

Circulation Research
Cardiovascular MedicinePhysiologyCardiology
2003English

Cellular Sources of Tissue Factor in Endotoxemia and Sepsis

Thrombosis Research
Hematology
2010English

Inhibition of Pulmonary Neutrophil Trafficking During Endotoxemia Is Dependent on the Stimulus for Migration

American Journal of Respiratory Cell and Molecular Biology
Respiratory MedicinePulmonaryClinical BiochemistryCell BiologyMolecular Biology
1999English

Inhibitory Role of the Acute Phase Proteins on Neutrophil Migration in Severe Sepsis

Critical Care
Critical CareIntensive Care Medicine
2009English

Complement 5a Receptor-Mediated Neutrophil Dysfunction Is Associated With a Poor Outcome in Sepsis

Cellular and Molecular Immunology
Infectious DiseasesMedicineAllergyImmunology
2015English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy